| [1]CAMPO E,SWERDLOW S H,HARRIS N L,et al.The 2008 WHO classification of lymphoid neoplasms and beyond:evolving concepts and practical applications [J].Blood,2011,117(19):5019-5032.
[2]ARBER D A,ORAZI A,HASSERJIAN R,et al.The 2016 revision to the World Health Organization classification of myeloid neoplasms and acute leukemia [J].Blood,2016,127(20):2391-2405.
[3]GROSCHEL S,SANDERS M A,HOOGENBOEZEM R,et al.A single oncogenic enhancer rearrangement causes concomitant EVI1 and GATA2 deregulation in leukemia [J].Cell,2014,157(2):369-381.
[4]KONOPLEV S,YIN C C,KORNBLAU S M,et al.Molecular characterization of de novo philadelphia chromosome-positive acute myeloid leukemia [J].Leuk Lymphoma,2013,54(1):138-144.
[5]NACHEVA E P,GRACE C D,BRAZMA D,et al.Does BCR/ABL1 positive acute myeloid leukaemia exist? [J].Br J Haematol,2013,161(4):541-550.
[6]MENDLER J H,MAHARRY K,RADMACHER M D,et al.RUNX1 mutations are associated with poor outcome in younger and older patients with cytogenetically normal acute myeloid leukemia and with distinct gene and MicroRNA expression signatures [J].J Clin Oncol,2012,30(25):3109-3118.
[7]FALINI B,MACIJEWSKI K,WEISS T,et al.Multilineage dysplasia has no impact on biologic,clinicopathologic,and prognostic features of AML with mutated nucleophosmin(NPM1) [J].Blood,2010,115(18):3776-3786.
[8]BACHER U,SCHNITTGER S,MACIJEWSKI K,et al.Multilineage dysplasia does not influence prognosis in CEBPA-mutated AML,supporting the WHO proposal to classify these patients as a unique entity [J].Blood,2012,119(20):4719-4722.
[9]SCHLENK R F,TASKESEN E,VAN NORDEN Y,et al.The value of allogeneic and autologous hematopoietic stem cell transplantation in prognostically favorable acute myeloid leukemia with double mutant CEBPA [J].Blood,2013,122(9):1576-1582.
[10]WALTER R B,OTHUS M,BURNETT A K,et al.Significance of FAB subclassification of "acute myeloid leukemia,NOS" in the 2008 WHO classification:analysis of 5848 newly diagnosed patients [J].Blood,2013,121(13):2424-2431.
[11]WANG S A,HASSERJIAN R P.Acute erythroleukemias,acute megakaryoblastic leukemias,and reactive mimics:a guide to a number of perplexing entities [J].Am J Clin Pathol,2015,144(1):44-60.
[12]SHIMIZU H,YOKOHAMA A,HATSUMI N,et al.Philadelphia chromosome-positive mixed phenotype acute leukemia in the imatinib era [J].Eur J Haematol,2014,93(4):297-301.
[13]DEN BOER M L,VAN SLEGTENHORST M,DE MENEZES R X,et al.A subtype of childhood acute lymphoblastic leukaemia with poor treatment outcome:a genome-wide classification study [J].Lancet Oncol,2009,10(2):125-134.
[14]CLAPPIER E,GRARDEL N,BAKKUS M,et al.IKZF1 deletion is an independent prognostic marker in childhood B-cell precursor acute lymphoblastic leukemia,and distinguishes patients benefiting from pulses during maintenance therapy:results of the EORTC children’s Leukemia group study 58951 [J].Leukemia,2015,29(11):2154-2161.
[15]BOER J M,MARCHANTE J R,EVANS W E,et al.BCR-ABL1-like cases in pediatric acute lymphoblastic leukemia:a comparison between DCOG/Erasmus MC and COG/St.Jude signatures [J].Haematologica,2015,100(9):e354-357.
[16]ROBERTS K G,MORIN R D,ZHANG J,et al.Genetic alterations activating kinase and cytokine receptor signaling in high-risk acute lymphoblastic leukemia [J].Cancer Cell,2012,22(2):153-166.
[17]HUNGER S P,MULLIGHAN C G.Redefining ALL classification:toward detecting high-risk ALL and implementing precision medicine [J].Blood,2015,125(26):3977-3987.
[18]ROBERTS K G,LI Y,PAYNE-TURNER D,et al.Targetable kinase-activating lesions in Ph-like acute lymphoblastic leukemia [J].N Engl J Med,2014,371(11):1005-1015.
[19]WESTON B W,HAYDEN M A,ROBERTS K G,et al.Tyrosine kinase inhibitor therapy induces remission in a patient with refractory EBF1-PDGFRB-positive acute lymphoblastic leukemia [J].J Clin Oncol,2013,31(25):e413-416.
[20]HARRISON C J,MOORMAN A V,SCHWAB C,et al.An international study of intrachromosomal amplification of chromosome 21(iAMP21):cytogenetic characterization and outcome [J].Leukemia,2014,28(5):1015-1021.
[21]PATRICK K,WADE R,GOULDEN N,et al.Outcome for children and young people with early T-cell precursor acute lymphoblastic leukaemia treated on a contemporary protocol,UKALL 2003 [J].Br J Haematol,2014,166(3):421-424.
[22]WOOD B L,WINTER S S,DUNSMORE K P,et al.T-Lymphoblastic Leukemia(T-ALL) shows excellent outcome,lack of significance of the Early Thymic Precursor(ETP) immunophenotype,and validation of the prognostic value of end-induction Minimal Residual Disease(MRD) in Children’s Oncology Group(COG) Study AALL0434 [J].Blood,2014,124(21):1.
[23]JAIN N,LAMB A V,O’BRIEN S,et al.Early T-cell precursor acute lymphoblastic leukemia/lymphoma(ETP-ALL/LBL) in adolescents and adults:a high-risk subtype [J].Blood,2016,127(15):1863-1869. |